Many people think of their veins as like the water pipes in their homes, which sometimes get clogged when “dirt” builds up. But in reality it is very different. In your arteries, and especially in your heart, the walls of the arteries interact with the blood that flows through them. And because this blood contains many different living cells, there is constant contact and interaction between them and the cells in the walls of your heart. For this reason, these cells are involved in the development of heart attacks.
The main cause of a heart attack is the buildup of plaque in the arteries called atherosclerosis. This collection does not happen suddenly; it grows slowly over many years, usually over a decade. Surprisingly, though, in most cases, it’s not the plaque itself that causes most heart attacks.
But why is this plate created? Part of the cause is inflammation. As you know, inflammation occurs when your body recognizes a wound or a foreign body. He immediately goes to work to repair the damage or remove the foreign body.
To understand the role of inflammation in the process, we need to look at how cholesterol is distributed throughout the body. We usually refer to this as LDL or HDL cholesterol, but these naturally refer to carrier cholesterol. (Cholesterol itself doesn’t dissolve in the blood, so it needs a carrier.) Many people have heard that LDL is the “bad cholesterol” and HDL is the “good cholesterol”, but this is not entirely true. Both types of cholesterol play important roles in your body.
The problem is that LDL enters and leaves the walls of the arteries. At low density, LDL lasts for a very short time, but at higher levels, more and more people get stuck, and as the concentration increases, so does the number stuck. When we take a closer look at LDL, let’s see why. LDL cells are not all the same size; some are big and some are small. And it’s the little ones that get stuck. This means that we want more of our ‘LDL’ to be larger; Unfortunately, we don’t have much control over this.
When large amounts of LDL get trapped, our immune system sees it as dangerous and sends out inflammatory cells to fix the problem. The first cells to respond to this process are called monocytes. They attach to the walls of the artery and soon begin to increase LDL concentration. As they grow, they turn into powerful agents called macrophages. The macrophages go after the LDL cells and begin to take over. Soon they are so full of LDL that they begin to foam; at this stage they are called foam cells.
At the same time, the immune system’s T cells are also working to remove LDL. They release so-called cytokines that speed up the process. Along with macrophages, they form what is known as adipose tissue which eventually develops as an inflammatory plaque on the arterial wall. This part of the plaque will continue to grow over the years, eventually forming a “hood.” Surprisingly, it is not this part of the inflamed plaque that causes most heart attacks. It can grow to the size of a coronary artery, but only 15% of heart attacks are caused by this blockage.
However, as plaque grows, inflammatory substances hidden by foam cells in plaque can weaken the lining. If this happens, a crack or channel will form inside, leading to the surface. The foam cells then release chemicals that can cause blood clots to form and it is this blood supply that completely blocks the artery. This is where most heart attacks occur.
What Makes This Possible?
Of course, many factors can contribute to this. And by avoiding them as much as possible, you can reduce your risk of having a heart attack. I am:
- Age (this is the one you can’t control). It usually takes years for plaque to form.
- Cholesterol level. High cholesterol triggers the process.
- Broom. The best way to keep LDL cholesterol low is to eat.
- Shoot. NO SMOKING.
- Arterial pressure. Keep your blood pressure low.
- obesity. Take care of your life.
- Little or no exercise. Make sure to train several times a week.
- Minimize stress.
- What happens when you go to the hospital?
Once you’re sure you’ve had a heart attack, it’s important to get to the hospital as soon as possible. One of the first things they’ll do there is prescribe medication to remove the blockage (if it doesn’t go away on its own). There are probably a number of experiments to perform as well. Two of them are ECG and blood pressure. The ECG is a device that monitors the electrical activity of the heart muscle; it emits a small electrical signal each time the heart beats. This will tell the cardiologist how well the heart is working and if any damage has occurred. A blood test is used to look for enzymes in the blood that have been released during a heart attack. They tell the doctor that a heart attack has happened.
Echocardiography will also be used to look directly at the heart. Uses ultrasonic waves; they are dispatched and beaten, and also show any damage that has occurred. An angiography will also be done (usually later in the surgery). In this case, the pigment is absorbed into the bloodstream and is examined using X-rays as it passes through the heart.
Once a blockage is found, the cardiologist inserts a catheter into a large vein in the groin. Using X-ray technology, the tube is guided through the closure of the coronary artery. The balloon is then used to dilate the artery. Then a stent (metal tube) is inserted to keep the vein open. Recent stalls have been sprayed to reduce the likelihood of frequent blockages.
In some cases, the use of a stent (or several stents) causes very serious damage and a bypass is necessary. In this case, the vein is removed from the foot and used as a passage. The blood then flows near the barrier.
Complications After a Heart Attack
When you leave the hospital, it is important to start an exercise program and someone can be created for you. Exercise strengthens the heart and reduces the risk of another heart attack. Depression and anxiety can also be a problem. According to one study, about 20% of heart attack victims were depressed or anxious shortly after leaving the hospital, and the remaining 20% suffered from mild depression after months of surgery. It can affect your recovery and it’s a good idea to try to avoid it. Stay positive and don’t let negative thoughts enter your mind. Stick to the things you love and be optimistic.